TonEBP suppresses IL-10-mediated immunomodulation

نویسندگان

  • Soo Youn Choi
  • Hwan Hee Lee
  • Jun Ho Lee
  • Byeong Jin Ye
  • Eun Jin Yoo
  • Hyun Je Kang
  • Gyu Won Jung
  • Seung Min An
  • Whaseon Lee-Kwon
  • Mario Chiong
  • Sergio Lavandero
  • Hyug Moo Kwon
چکیده

TonEBP is a key transcriptional activator of M1 phenotype in macrophage, and its high expression is associated with many inflammatory diseases. During the progression of the inflammatory responses, the M1 to M2 phenotypic switch enables the dual role of macrophages in controlling the initiation and resolution of inflammation. Here we report that in human and mouse M1 macrophages TonEBP suppresses IL-10 expression and M2 phenotype. TonEBP knockdown promoted the transcription of the IL-10 gene by enhancing chromatin accessibility and Sp1 recruitment to its promoter. The enhanced expression of M2 genes by TonEBP knockdown was abrogated by antagonism of IL-10 by either neutralizing antibodies or siRNA-mediated silencing. In addition, pharmacological suppression of TonEBP leads to similar upregulation of IL-10 and M2 genes. Thus, TonEBP suppresses M2 phenotype via downregulation of the IL-10 in M1 macrophages.

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عنوان ژورنال:

دوره 6  شماره 

صفحات  -

تاریخ انتشار 2016